Avoiding Common Pediatric Errors (Avoiding Common Errors) by Anthony D. Slonim MD DrPH

By Anthony D. Slonim MD DrPH

This notebook succinctly describes 250 blunders generally made through physicians taking good care of youngsters in all scientific settings and provides useful, easy-to-remember information for heading off those error. effortless to learn in the course of a brief rotation, the publication identifies the entire key pitfalls in info accumulating, interpretation, and scientific choice making. each one mistakes is defined in a brief, clinically suitable vignette, by means of an inventory of items that are supposed to continually or by no means be performed in that context and tips to stay away from or ameliorate difficulties. assurance contains all parts validated at the American Board of Pediatrics certification and recertification checks.

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Avoiding Common Pediatric Errors (Avoiding Common Errors)

This notebook succinctly describes 250 error generally made through physicians taking care of childrens in all scientific settings and provides sensible, easy-to-remember information for averting those error. effortless to learn in the course of a quick rotation, the e-book identifies all of the key pitfalls in information collecting, interpretation, and scientific choice making.

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Extra resources for Avoiding Common Pediatric Errors (Avoiding Common Errors)

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In most cases, this jaundice appears between the second and fifth days of life and clears with time, often without treatment. Also, once this type of jaundice disappears, there is no evidence that it will appear again or that it has any lasting effects on the baby. Physiologic Jaundice Physiologic jaundice in healthy term newborns follows a typical pattern. The average total serum bilirubin level usually peaks at 5 to 6 mg/dL (86–103 µmol/L) on the third to fourth day of life and declines over the first week after birth.

Abnormalities that result in desaturated blood bypassing the alveolus include an intracardiac right-to-left shunt or an intrapulmonary shunts. Intracardiac shunts may result from either from cyanotic congenital heart disease (CHD) or pulmonary hypertension resulting in a right-to-left shunt at the ductal or intracardiac level. Intrapulmonary shunts can arise from an arteriovenous malformation or fistula, for example. The clinician most frequently is faced with distinguishing whether cyanosis is a result of pulmonary or cardiac disease but should remember that other less causes of cyanosis also exist.

In contrast, when the cyanosis results from lung disease at the capillary level, an increased alveolar O2 content will result in increased diffusion across the alveolar-capillary interface, thereby increasing arterial pO2. Pulmonary hypertension with a patent ductus can complicate the test because the cyanosis in this setting is created by right-to-left shunt across the patent ductus arteriosus (external to the lung). With the application of 100% O2, there can be some diminishment in the pulmonary hypertension, which both increases the amount of blood going to the lungs and diminishes the ductal rightto left shunt.

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